142. Roche, D. J., King, A. C., Cohoon, A. J., & Lovallo, W. R. (2013). Hormonal contraceptive use diminishes salivary cortisol response to psychosocial stress and naltrexone in healthy women. Pharmacology Biochemistry and Behavior, 109, 84–90. doi:10.1016/j.pbb.2013.05.007.
143. Kirschbaum, C., Platte, P., Pirke, K., & Hellhammer, D. (1996). Adrenocortical activation following stressful exercise: Further evidence for attenuated free cortisol responses in women using oral contraceptives. Stress and Health, 12(3), 137–143. doi:doi:10.1002/(SICI)1099-1700(199607)12:3<137::AID-SMI685>3.0.CO;2-C; Roche, D. J., King, A. C., Cohoon, A. J., & Lovallo, W. R. (2013). Hormonal contraceptive use diminishes salivary cortisol response to psychosocial stress and naltrexone in healthy women. Pharmacology Biochemistry and Behavior, 109, 84–90. doi:10.1016/j.pbb.2013.05.007; Bonen, A., Haynes, F. W., & Graham, T. E. (1991). Substrate and hormonal responses to exercise in women using oral contraceptives. Journal of Applied Physiology, 70(5), 1917–1927. doi:10.1152/jappl.1991.70.5.1917; Kirschbaum, C., Pirke, K., & Hellhammer, D. H. (1995). Preliminary evidence for reduced cortisol responsivity to psychological stress in women using oral contraceptive medication. Psychoneuroendocrinology, 20(5), 509–514. doi:10.1016/0306-4530(94)00078-o; Meulenberg, P., Ross, H., Swinkels, L., & Benraad, T. (1987). The effect of oral contraceptives on plasma-free and salivary cortisol and cortisone. Clinica Chimica Acta, 165(2-3), 379–385. doi:10.1016/0009-8981(87)90183-5; Meulenberg, P., & Hofman, J. (1990). The effect of oral contraceptive use and pregnancy on the daily rhythm of cortisol and cortisone. Clinica Chimica Acta, 190(3), 211–221. doi:10.1016/0009-8981(90)90175-r; Crewther, B. T., Hamilton, D., Casto, K. et al. (2015). Effects of oral contraceptive use on the salivary testosterone and cortisol responses to training sessions and competitions in elite women athletes. Physiology and Behavior, 147, 84–90. doi:10.1016/j.physbeh.2015.04.017.
144. Hertel, J., König, J., Homuth, G. et al. (2017). Evidence for stress-like alterations in the HPA-axis in women taking oral contraceptives. Scientific Reports, 7(1), 1–14. doi:10.1038/s41598-017-13927-7.
145. Simunková, K., Stárka, L., Hill, M. et al. (2008). Comparison of total and salivary cortisol in a low-dose ACTH (Synacthen) test: Influence of three-month oral contraceptives administration to healthy women. Physiological Research, 57, S193–S199; Wiegratz, I., Jung-Hoffmann, C., & Kuhl, H. (1995). Effect of two oral contraceptives containing ethinylestradiol and gestodene or norgestimate upon androgen parameters and serum binding proteins. Contraception, 51(6), 341–346. doi:10.1016/0010-7824(95)00098-u.
146. Jacobs, A. J., Odom, M. J., Word, R. A., & Carr, B. R. (1989). Effect of oral contraceptives on adrenocorticotropin and growth hormone secretion following CRH and GHRH administration. Contraception, 40(6), 691–699. doi:10.1016/0010-7824(89)90072-3.
147. Kirschbaum, C., Kudielka, B. M., Gaab, J. et al. (1999). Impact of gender, menstrual cycle phase, and oral contraceptives on the activity of the hypothalamus-pituitary-adrenal axis. Psychosomatic Medicine, 61(2), 154–162. doi:10.1097/00006842-199903000-00006.
148. Gaffey, A. E., Wirth, M. M., Hoks, R. M. et al. (2014). Circulating cortisol levels after exogenous cortisol administration are higher in women using hormonal contraceptives: Data from two preliminary studies. Stress, 17(4), 314–320. doi:10.3109/10253890.2014.919447.
149. Kirschbaum, C., Kudielka, B. M., Gaab, J. et al. (1999). Impact of gender, menstrual cycle phase, and oral contraceptives on the activity of the hypothalamus-pituitary-adrenal axis. Psychosomatic Medicine, 61(2), 154–162. doi:10.1097/00006842-199903000-00006.
150. Jacobs, A. J., Odom, M. J., Word, R. A., & Carr, B. R. (1989). Effect of oral contraceptives on adrenocorticotropin and growth hormone secretion following CRH and GHRH administration. Contraception, 40(6), 691–699. doi:10.1016/0010-7824(89)90072-3.
151. Simunkovа, K., Stаrka, L., Hill, M. et al. (2008). Comparison of total and salivary cortisol in a low-dose ACTH (Synacthen) test: Influence of three-month oral contraceptives administration to healthy women. Physiological Research, 57, S193–S199.
152. Hertel, J., König, J., Homuth, G. et al. (2017). Evidence for stress-like alterations in the HPA-axis in women taking oral contraceptives. Scientific Reports, 7(1), 1–14. doi:10.1038/s41598-017-13927-7.
153. Hertel, J., König, J., Homuth, G. et al. (2017). Evidence for stress-like alterations in the HPA-axis in women taking oral contraceptives. Scientific Reports, 7(1), 1–14. doi:10.1038/s41598-017-13927-7.
154. Conrad, C. D. (2008). Chronic stress-induced hippocampal vulnerability: The glucocorticoid vulnerability hypothesis. Reviews in the Neurosciences, 19(6). doi:10.1515/revneuro.2008.19.6.395; Magarinos, A. M., Verdugo, J. M., & McEwen, B. S. (1997). Chronic stress alters synaptic terminal structure in hippocampus. Proceedings of the National Academy of Sciences, 94(25), 14002–14008. doi:10.1073/pnas.94.25.14002.
155. Klengel, T., Mehta, D., Anacker, C. et al. (2013). Allele-specific FKBP5 DNA demethylation mediates gene–childhood trauma interactions. Nature Neuroscience, 16, 33–41. doi:10.1038/nn.3275.
156. Hertel, J., König, J., Homuth, G. et al. (2017). Evidence for stress-like alterations in the HPA-axis in women taking oral contraceptives. Scientific Reports, 7(1), 1–14. doi:10.1038/s41598-017-13927-7.
